Anavar 50mg Tablets | 100-Count
Oxandrolone · 322:24 · 2-oxa ring
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≥99% Pure · COA
Both Anavar and Winstrol are DHT-derived oral AAS that do not aromatize. Both have high anabolic:androgenic selectivity relative to testosterone. Yet their structural modifications — Anavar's 2-oxa ring vs Winstrol's pyrazole ring — produce meaningfully different receptor pharmacology, SHBG dynamics, and research applications.
Shop All QSC Steroids →| Property | Anavar (Oxandrolone) | Winstrol (Stanozolol) |
|---|---|---|
| Parent | DHT (2-oxa modification) | DHT (pyrazole A-ring fusion) |
| Anabolic:Androgenic | 322:24 | 320:30 |
| Key Structural Feature | 2-oxa ring (O replaces C2) | Pyrazole ring (unique in AAS class) |
| Aromatization | None | None |
| SHBG Binding | Low affinity for SHBG | High affinity — displaces other steroids |
| Injectable Available | No | Yes (50mg/mL aqueous) |
| Hepatic Excretion | Predominantly renal (unique) | Predominantly hepatic |
| Half-Life | ~9 hours (oral) | ~9 hrs (oral), ~24 hrs (injectable) |
| Hepatotoxicity | Relatively low (renal excretion) | Moderate (hepatic) |
| QSC Price | $190 (50mg 100-count) | $130–160 (100-count), $270 (inj.) |
Anavar (2-oxa): An oxygen atom replaces the C2 carbon in the A-ring. This creates oxolane (cyclic ether) chemistry, changes the ring electronics, and produces a compound with tissue-selective AR transactivation — particularly strong in skeletal muscle with weaker activity in androgenic tissues.
Winstrol (pyrazole): A pyrazole ring is fused at the 2,3-position of the A-ring — a modification seen in no other AAS. This nitrogen-containing heterocycle confers completely different binding geometry at the AR ligand-binding domain.
This is the key functional difference for multi-compound research protocols. Stanozolol (Winstrol) has unusually high affinity for SHBG — it competitively displaces other steroids from albumin binding, increasing the free fraction of co-administered AAS. Oxandrolone (Anavar) has low SHBG affinity and does not produce this displacement effect. In research studying free vs bound steroid activity, this distinction is fundamental.
Anavar is predominantly excreted renally — ~28% unchanged in urine — rather than hepatically. This is unusual for a C17α-alkylated oral steroid and explains its lower hepatotoxicity relative to Winstrol and other 17α-AA. For hepatic toxicity research comparing oral AAS, Anavar vs Winstrol provides a useful within-class contrast.
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